CALL FOR PAPERS Cardiovascular and Cerebrovascular Aging–New Mechanisms and Insights Cardiac aging is initiated by matrix metalloproteinase-9-mediated endothelial dysfunction

نویسندگان

  • Andriy Yabluchanskiy
  • Yonggang Ma
  • Ying Ann Chiao
  • Elizabeth F. Lopez
  • Andrew P. Voorhees
  • Hiroe Toba
  • Michael E. Hall
  • Hai-Chao Han
  • Merry L. Lindsey
  • Yu-Fang Jin
چکیده

Andriy Yabluchanskiy, Yonggang Ma, Ying Ann Chiao, Elizabeth F. Lopez, Andrew P. Voorhees, Hiroe Toba, Michael E. Hall, Hai-Chao Han, Merry L. Lindsey, and Yu-Fang Jin San Antonio Cardiovascular Proteomics Center, San Antonio, Texas; Department of Physiology and Biophysics, Mississippi Center for Heart Research, University of Mississippi Medical Center, Jackson, Mississippi; Department of Pathology, University of Washington, Seattle, Washington; Department of Mechanical Engineering, University of Texas at San Antonio, San Antonio, Texas; Division of Pathological Sciences, Department of Clinical Pharmacology, Kyoto Pharmaceutical University, Kyoto, Japan; Division of Cardiology, University of Mississippi Medical Center; Jackson, Mississippi; Research Service, G.V. (Sonny) Montgomery Veterans Affairs Medical Center; Jackson, Mississippi; and Department of Electrical and Computer Engineering, University of Texas at San Antonio, San Antonio, Texas

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Cardiac aging is initiated by matrix metalloproteinase-9-mediated endothelial dysfunction.

Aging is linked to increased matrix metalloproteinase-9 (MMP-9) expression and extracellular matrix turnover, as well as a decline in function of the left ventricle (LV). Previously, we demonstrated that C57BL/6J wild-type (WT) mice > 18 mo of age show impaired diastolic function, which was attenuated by MMP-9 deletion. To evaluate mechanisms that initiate the development of cardiac dysfunction...

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تاریخ انتشار 2014